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Selected References

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After a penetrating lesion in the central nervous system, astrocytes enlarge, divide, and participate in creating an environment that adversely affects neuronal regeneration. We have recently shown that the neural cell adhesion molecule (N-CAM) partially inhibits the division of early postnatal rat astrocytes in vitro. Animals have been lesioned in the cerebral cortex, hippocampus, or striatum with a Hamilton syringe and needle at defined stereotaxic positions. On one facet, the lesions were concomitantly infused with N-CAM or with one of many N-CAM-related molecules. As a management, a peptide of the identical composition as the N-CAM decapeptide but of random sequence was infused on the contralateral aspect of the brain. The inhibition was greatest in the cortical lesion websites (approximately 50%) and was much less pronounced within the hippocampus (approximately 25%) and striatum (approximately 20%). Two weeks after the lesion, the cerebral cortical sites infused with N-CAM continued to exhibit a significantly smaller inhabitants of dividing astrocytes than the websites on the alternative facet.

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Del Rio JA, Soriano E. Immunocytochemical detection of 5′-bromodeoxyuridine incorporation within the central nervous system of the mouse. Giulian D, Chen J, Ingeman JE, George JK, Noponen M. The position of mononuclear phagocytes in wound healing after traumatic harm to adult mammalian brain. Smith GM, Rutishauser U, Silver J, Miller RH. Maturation of astrocytes in vitro alters the extent and molecular foundation of neurite outgrowth. Full text is available as a scanned copy of the original print model. Get a printable copy of the complete article (1.6M), or click on on a web page picture under to browse page by page. Links to PubMed are additionally obtainable for Selected References. Immunohistological localization of the adhesion molecules L1, N-CAM, and MAG in the creating and adult optic nerve of mice.